Fig. 8From: Gut-dependent microbial translocation induces inflammation and cardiovascular events after ST-elevation myocardial infarctionSchematic illustration of the proposed interaction between STEMI, microbial translocation, and future ischemic events. Increased gut permeability and bacterial translocation from the gut lumen due to intestinal hypoperfusion post-STEMI are involved in the pathogenesis of monocytosis and inflammation and have the potential of contributing to further ischemic events and heart failureBack to article page